A drug that blocks glutamate overactivation via the glutamate receptor 'mGluR5' was used in the study.

一种通过谷氨酸受体’mGluR5’阻断谷氨酸过于活跃的药物正在研究中使用。

Howlett found the fruit fly's metabotropic glutamate receptor (DmGluRA) gene, when mutated, increased the excitability of the neuron by preventing PI3K from doing its job.

赫尔特发现当果蝇的亲代谢性谷氨酸盐受体基因发生突变时，将通过阻止PI3K的作用来增加神经元的兴奋性。

More precisely, they block the action of a form of glutamate receptor known as the NMDA receptor, which plays a critical role in brain development, learning, memory and neural processing in general.

说得更精确些，这些药物阻断了一种叫做NMDA型的麸胺酸受体，该受体对于大脑的发育、习、忆以及一般的神经活动都扮演著举足轻重的角色。

A mutation in a glutamate receptor gene common to both the fruit fly and humans has the ability to disrupt that regulatory mechanism.

共同存在于果蝇和人类的谷氨酸受体基因的突变可以破坏这种调节机制。

Further pharmacological experiments revealed that S100B secretion was critically dependent on presynaptic release of neurotransmitter and activation of metabotropic glutamate receptor 3.

进一步哟来实验揭示了S100B释放是一种突触前神经递质释放的方式，并且依赖于代谢型谷氨酸受体3的激活。

Objective To explore the expression of glutamate receptor interacting protein (GRIP) after ischemia-reperfusion in the rat brain.

目的探讨脑缺血再灌注大鼠半暗带皮质中谷氨酸受体相互作用蛋白（GRIP）表达变化的规律。

Glutamate receptor (GluR) is widespread in the central nervous system, and plays a significance role in the excitary neurotransmission.

谷氨酸受体在中枢神经系统分布广泛，其在介导兴奋性神经递质信号传递过程中起重要作用。

Distribution of the metabotropic glutamate receptor 2 and 3 subtypes-like immunoreactive(mGluR2/3-LI)neuronal cell bodies and fibers was immunohistochemically examined in the rat brain.

用免疫组化技术观察了代谢型谷氨酸受体2和3亚型阳性神经元胞体和纤维在大鼠脑内的定位分布。

Conclusion: NO may play a role in the pathological process of schizophrenia caused by abnormal glutamate NMDA receptor function.

结论：NO可能在谷氨酸NMDA受体功能异常引起的精神分裂症的病理过程发挥一定作用。

Objective To evaluate the effect of glutamate on adenosine receptor A2A regulating activity of nitric oxide synthase (NOS) in microglia.

目的探讨谷氨酸是否能够影响腺苷A2A受体对一氧化氮合酶（NOS）活力的调节。

Ketamine blocks the activity of glutamate by occupying the receptor molecules in the brain to which it would otherwise bind.

克他命药剂能够通过占据大脑中的受体分子而阻止谷胺酸酯传递素的行为活动,否则这些受体分子就会彼此相连.

In the patient and the animal model of hepatic encephalopathy, the function of glutamate neurotransmitter is bad, and receptor binding site is reductive and even lack.

肝性脑病患者及动物模型中谷氨酸神经递质功能不良，其受体结合位点减少甚至缺失。

The results suggested that glutamate mechanism (mediated by NMDA receptor) is involved in the modulation of the electric activities of ARC neurons.

结果提示：下丘脑ARC神经元的电活动有谷氨酸机制（介导于NMDA受体）的参与。

In this study we have investigated the possibility of glutamate release through volume sensitive channels upon receptor stimulation.

本研究探讨了在受体激活状态下谷氨酸通过体积敏感通道释放的机制。

Conclusion High level of glutamate can reverse the effect of A2A receptor antagonist inhibiting increase of activity of NOS in nervous system.

结论高浓度谷氨酸可逆转腺苷A2A受体激动剂抑制升高NOS活力的作用。

Radioactive ligand-receptor binding test was performed with glutamate as the ligand with 3H labeling.

并以3H标记的谷氨酸为配基进行放射性配基-受体结合实验。

The receptor cells have different receptors on their surface where the glutamate is taken in to activate the cell.

受体细胞在表层上有不同的受体，吸入谷氨酸使细胞活跃起来。

Conclusion The excitability and the open probability of NMDA receptor channel of hypothalamic neurons increased by hypoxia is related to glutamate.

结论缺氧及谷氨酸能提高下丘脑神经元NMDA通道的兴奋性，促进NMDA通道开放增加。